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Classical homocystinuria has been recognised as a protein misfolding disorder as many of the known pathogenic mutations result in aggregation and degradation of the CBS enzyme 9,10

The rest of the binding sites non-specifically interacted with other slightly charged proteins, such as histidine-rich protein, leading to impurities later on

Recent findings indicate that BCP alleviates oxidative stress, inflammation, and apoptosis, demonstrating a protective effect against doxorubicin-induced cardiotoxicity (Meeran et al., 2019)

However, NAC appears to have a cell type-specific action: in human bronchial epithelial cells, NAC inhibited silica-induced NFkB at a concentration of 5 mM [60]