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Studies demonstrate that the intracellular portion of CFTR binds to protein modules referred to as PSD-95/Discs-large/ZO-1 (PDZ) domains, which promote proteinprotein interactions

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reported that inhibition of the Xc- transport system using sulfasalazine, while ineffective as a monotherapy, was highly effective to eliminate AML cell lines when combined with Buthionine sulfoximine (BSO), an inhibitor of glutamyl cysteine synthetase, the first enzyme in the synthesis of GSH

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