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These therapeutics irreversibly inhibit EGFR kinase activity by forming a covalent bond with Cys797 in the ATP-binding pocket and consequently overcoming resistance to reversible TKIs in lung cancer patients with the acquired T790M/L858R EGFR mutation [212, 213]

Increased ROS levels induce HK II expression and make cancer cells sensitive to 3-BP treatment, and thereby promote cell death via ROS-mediated apoptosis (the black box indicates monocarboxylate transporter-1, and the white box indicates monocarboxylate transporter-4)

Adult neurobehavioral alterations in male and female mice following developmental exposure to paracetamol (acetaminophen): Characterization of a critical period

Draft genome sequences of Bifidobacterium angulatum GT102 and Bifidobacterium adolescentis 150: focusing on the genes potentially involved in the gut-brain axis

10.1523/JNEUROSCI.5369-03.2004 J