Vol. XVIII · Free shipping $75+ · Read the collection
Feature · Product Review
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Description

It was reported that it could raise intracellular GSH by increasing SLC7A11 expression, efficiently leading to GPX4 enhancement and ferroptosis suppression in the ISO-induced injury model of H9C2 cells and rat myocardium [49]

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Griefahn (2001)

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An NAD+-dependent transcriptional program governs self-renewal and radiation resistance in glioblastoma

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