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doi: 10.1111/j.1468-1331.2009.02748.x

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176 Collectively, these advances underscore a fundamental conceptual shift: proteostasis must now be viewed not as a collection of isolated degradation mechanisms but as a coherent, adaptive, and deeply integrated cellular defense network whose resilience determines disease trajectory

Inhibition of the immunoproteasome LMP2 ameliorates ischemia/hypoxia-induced bloodbrain barrier injury through the Wnt/-catenin signalling pathway