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Mechanisms behind the pathology of the disease are not fully understood, however, previous studies indicate that certain strains of bacteria residing in the intestines may contribute to the disease

doi:10.1177/2398212819858249

In oxaliplatin-induced CIPN models, melatonin prevented the loss of mitochondrial membrane potential (m), promoted nerve regeneration in oxaliplatin-challenged Neuro-2a cells and prevented the loss of nitrosation of proteins and antioxidant enzymes by improving nitrooxidative stress mediated by the chemotherapeutic agent oxaliplatin [45] without interfering with oxaliplatin cytotoxic activity in human colon cancer cell lines

Korshunov VA, Berk BC Arteriosclerosis, thrombosis, and vascular biology

Coenzyme Q10 may reduce the anticoagulant effect of warfarin