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Description

TLR-driven early glycolytic reprogramming via the kinases TBK1-iKK supports the anabolic demands of dendritic cell activation

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In fact, ALS and FTD represent two opposite ends of the same disease spectrum, with overlapping clinical symptoms and genetics (Shahheydari et al., 2017)

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10.1038/s41392-021-00701-5 83 ZhangT.ChaboryE.BritanA.GrignardE.PitiotO.SaezF.et al (2008)

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1 2 Neurochemical Research

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PinX1 depletion improves liver injury in a mouse model of nonalcoholic fatty liver disease via increasing telomerase activity and inhibiting apoptosis

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