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Beta-catenin deletion in hepatoblasts disrupts hepatic morphogenesis and survival during mouse development

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doi: 10.1101/gr.2332504 120 TabeL

Turell L, Radi R, Alvarez B

As previously described, perinatal dietary transitions often induce ruminal dysbiosis, which compromises glucogenic precursor supply and promotes extensive fat mobilization, thereby leading to excessive ketone body production in the liver