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The fading of the observed initial growth inhibition is likely due to the lack of additional T-cell co-stimulation, the limited lifespan of the co-inoculated PBMCs and the absence of T-cell influx into the developing tumours, as would normally occur in an immunocompetent host

Levofloxacin persisters (levopersisters) were quantified as CFU/mL, and minimum inhibitory concentrations (MICs) were assessed using the broth microdilution method, following the Clinical and Laboratory Standards Institute (CLSI) guidelines [21]

438 Exosomes are formed through the inward budding of the endosomal membrane, resulting in the pinching off and release of intraluminal vesicles (ILVs) inside endosomes

A pilot study of cerebral dialysate from 26 ICU patients with TBI defined two clinically relevant metabolic states based on distinct changes in glucose, lactate and pyruvate as well as metabolites related to ketone metabolism [46]

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