In TMZ-resistant GBM cells (such as A172-R and T98G-R cells), boric acid treatment significantly increased the expression level of NCOA4, elevated intracellular iron levels, and triggered lipid peroxidation and GSH depletion, thereby promoting ferritin autophagy and inducing ferroptosis ( - is blocked (erastin treatment), thereby inducing ferroptosis ( Radiation therapy can induce lipid peroxidation by generating reactive oxygen species (ROS), which are key triggers of ferroptosis
Therefore, the increased expression of miR-153-3p inhibited neuronal cell apoptosis and decreased inflammation in microglia by blocking the NF-B/iNOS/COX2 pathway while enhancing activation of the Nrf2/HO-1 signaling cascade
Cell-Free Protein Synthesis System Prepared from Insect Cells by Freeze-Thawing
Comparison of tirzepatide and dulaglutide on major adverse cardiovascular events in participants with type 2 diabetes and atherosclerotic cardiovascular disease: SURPASS-CVOT design and baseline characteristics
Based on this systematic review of the literature, this article summarizes current advances in the modulation of inflammationoxidative stress networks by natural products and elucidates their compoundtargetpathway interactions, thus providing theoretical foundations and innovative therapeutic perspectives for the treatment of viral pneumonia (Table 1
