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The administration of NAC up to 2 h after the brain injury resulted in an improvement in the activity of mitochondrial complexes, enhanced oxidative phosphorylation, and a reduction in the rate of ROS production [11]

Huang SY, Dong ZS, Chen ZH, et al

In the nucleus, protein phosphatase 2 A (PP2A)-mediated dephosphorylation at serine 298 residue affects the interaction of the nuclear localization signal of HDAC4 and MEF2, finally altering the neuronal activity

Polymorphisms arise from the occurrence of gene conversion events, deletions, duplications, multiple nucleotide insertions/deletions, and SNPs

It protects neuronal cells by mitigating oxidative stress, reducing inflammation, and enhancing mitochondrial function, thereby alleviating cerebrovascular injury and slowing the progression of VaD