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Description

Tops initiated partly the cell recovery following the cell mortality by tert -butyl hydroperoxide, which showed other relevant mechanisms rather than PI3K/Akt and HO-1 were responsible for Tops initiated cell recovery[]

Emerging evidence implicates a key role for non-mutational drug resistance mechanisms underlying the survival of residual cancer persister cells 2,3,4

TC2N has been demonstrated to activate the Wnt signalling pathway, thus promoting the invasive metastasis of HCC cells

ATF4 transcription plays a key role in maintaining cellular homeostasis, thereby promoting cellular adaptation

GSTs are divided into 14 classes: phi (GSTF), tau (GSTU), theta (GSTT), zeta (GSTZ), lambda (GSTL), metaxin, hemerythrin (GSTH), iota (GSTI), glutathione-dependent dehydroascorbate reductase (DHAR), tetrachloro hydroquinone dehalogenase (TCHQD), -subunit classes of the eukaryotic translation elongation factor 1B (EF1B), glutathionyl hydroquinone reductase (GHR), GSTs with two thioredoxins (GST2N), and microsomal prostaglandin E synthase type 2 (mPGES2) 2,3