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Abstract Background Oral melanoacanthoma (OM) is a rare, reactive, and benign proliferation of two cell types: keratinocytes and melanocytes

Proper laboratory handling involves using sterile techniques to prevent contamination during reconstitution, as these peptides are sensitive to environmental factors

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In this study, we have assessed disulfirams effects on neuroblastoma using in vitro and in vivo models, focusing on how it modulates the neuroblast transcriptome and epigenome, particularly via MYCN and KAT2A