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It can promote the proliferation of anaplastic thyroid cancer through inducing H4K12 Kla, mediating driver gene transcription, and cell cycle dysregulation (93)

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Conclusion: Given the highly potent pan-genotypic antiviral activity of BEM and RZR, the high resistance barrier of BEM, the complementary mechanisms of action of BEM and RZR, the clinically demonstrated safety and efficacy of each agent when administered individually, and the lack of drug-drug interaction( DDI) between BEM and RZR, combination treatment with these two agents for HCV infections is highly attractive and is currently being evaluated in a Phase 2 clinical trial

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P.BourdichonF.FanningS.et al (2013)