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OTHER REGULATORS OF THE PENTOSE PHOSPHATE PATHWAY It has been shown that the activation of mammalian target of rapamycin complex 1 (mTORC1) increases the metabolic flux through both glycolysis and the oxidative arm of the pentose phosphate pathway ( In contrast, the non-oxidative arm of the pentose phosphate pathway is enhanced in the presence of oncogenic Kras (Kras G12D ) in pancreatic tumors (Ying et al., 2012)

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(94) revealed that M2-derived exosomes suppress HCS activation by inhibiting the direct target of miRNA-411-5p , Calmodulin-Regulated Spectrin-Associated Protein 1 ( CAMSAP1 ), during the transition of NASH to NAFL

Akinrotimi O, Riessen R, VanDuyne P, Park JE, Lee YK, Wong LJ, et al