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This paper reviews the Met metabolism in normal conditions and its dysregulation under abnormal conditions, the possible causes of hypermethioninemia, and its connection to BA pathogenesis: Abnormal hepatic level of Met could lead to a perturbation of redox homeostasis and mitochondrial functions of hepatocytes, enhancement of viral infectivity, and dysregulation of innate and adaptative immune cells in response to infection/damage of the liver contributing to the initiation/progression of BA

Edaravone attenuates the proinflammatory response in amyloid-B-treated microglia by inhibiting nlrp3 inflammasome-mediated il-1 Secretion

USA 118 , e2112560118 (2021)

Wolrab D, Chocholoukov M, Jirsko R, Peterka O, Holapek M

After blocking, membranes were incubated overnight at 4 C with the primary antibodies listed in Table 1