For GOF mutations, developing targeted therapies that specifically inhibit the oncogenic functions of GOF mutant p53 may help suppress the survival and progression of tumors
On the other hand, EDV upregulates the expression of genes involved in neurotransmitter turnover, adenosine A1 receptor (Adora1), solute carrier family 6 member 15 (Slc6a15), catechol-O-methyltransferase (Comt)
Prez S, Talns-Visconti R, Rius-Prez S, Finamor I, Sastre J (2017) Redox signaling in the gastrointestinal tract
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Regulative potential of glutaminerelation to glutathione metabolism
