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Clinical reports suggest broader applications

An autoimmune disorder characterized by severe thrombocytopenia secondary to immune destruction of platelets that is responsible for increased bleeding risk

The resulting GABA then potentially affects the brain via the vagus nerve via the gut-brain axis or by increasing GABA levels in the blood

Molecular Basis of G6PD Deficiency In principle, genetically determined deficiency of G6PD, like that of any other protein, might be attributed either to quantitative changes, such as mutations that affect the amount of the enzyme but not its structure, or to qualitative changes, such as mutations that affect the structure of the enzyme and hence its stability or catalytic efficiency

Some peptides are FDA-approved drugs