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These novel findings are complemented by the demonstration of reduced GPx1 in the colon of mice with DSS-induced colitis [41, 42], murine colitis induced by knockout of GPx1 and GPx2 [43], and genome-wide association studies that note polymorphisms in antioxidant genes, including GPxs in IBD [44, 45]

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C/EBP deficiency in podocytes has been shown to aggravate podocyte senescence and kidney damage through the AMPK/mTOR pathway (mechanisms associated with metabolism) in aging mice