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Furthermore, while enzymatic removal of the L585 residue decreased FcRn binding for fusion designs with WT albumin, this was not the case for the corresponding QMP-engineered variants

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Neurobiol Learnand Mem 96: 564582

Front Oncol (2022) 12:802009

Dual-specificity phosphatase 26 protects against nonalcoholic fatty liver disease in mice through transforming growth factor beta-activated kinase 1 suppression