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The compound appears to work by increasing intracellular NAD+ levels (through preventing nicotinamide consumption by NNMT), which activates sirtuins and enhances mitochondrial function and fatty acid oxidation, while also promoting a phenomenon called "browning" of white adipose tissue - the transformation of energy-storing white fat cells into metabolically active beige/brown adipocytes that burn fatty acids for thermogenesis rather than storing them

The observations that CRPC is greatly enriched in cells with the AR - BCL-2 + phenotype, which characterizes normal prostatic basal cells, are fully consistent with our recent findings that ARPIs induce PCa plasticity by concurrently upregulating PCa cells with basal, mesenchymal, neural and stem cell gene expression signatures

Reported by Dustin Newman, Laboratory Director Approved by Tori Johnson, Operations Manager Vanguard Laboratory, Olympia WA July 18, 2025

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