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Description

Thus, exploring the effects of different ubiquitin-specific enzymes on ferroptosis in tumor cells is a new research direction

Dysregulated mTORC1 renders cells critically dependent on desaturated lipids for survival under tumor-like stress

Crossovers (COs) between homologues create the physical connections that enable their biorientation on the meiotic spindle 1 , yet the very same DNA exchanges are suppressed in somatic cells because they can provoke deleterious chromosomal rearrangements and loss-of-heterozygosity 2