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The frequencies presented, therefore, represent the proportion of the 3525 individuals exposed to clomipramine who experienced an event of the type cited on at least one occasion while receiving clomipramine

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AraC resistance in TP53 m ut AML requires the mevalonate pathway byproduct, GGPP m ut To determine the specific mevalonate pathway byproducts necessary for mitochondria-dependent chemoresistance, we performed rescue experiments in the representative TP53 mut AML clone, M14-Mut1, treated with a rescue agent and rosuvastatin for 48 h with AraC added for the last 24 h (Schema summarizing targets in Fig