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Toxicity of monosodium glutamate intake on different tissues induced oxidative stress: a review

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First, it inhibits ferroptosis by upregulating iron metabolism genes, including FTH1, SLC40A1, HMOX1 and MT1G

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39,40 This observation suggests the presence of a significant residual cardiovascular risk, that is, the component of an individual patients cardiovascular risk that is not influenced by existing treatments