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At the immune level, metabolic crosstalk critically shapes polarization: persistent long-chain fatty acid (LCFA) exposure depletes intestinal SCFAs while promoting differentiation of nave T cells into pro-inflammatory Th1/Th17 subsets in the small intestine (Haghikia et al., 2015)

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found that animals with multiple sclerosis had significantly lower levels of catalase in the hippocampus, but no changes were observed in plasma

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The brain on drugs: from reward to addiction