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Furthermore, -sitosterol increases the expression of death receptors and blocks the inhibitory effect of IAP proteins on caspases, making cancer cells more sensitive to exogenous death signals (84)

2) [40], which is elevated following SeNP exposure, and ultimately selenide (H2Se) [68]

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Efficacy of glutathione for the treatment of non-alcoholic fatty liver disease: an open-label, single-arm, multicenter, pilot study