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Indeed, during APAP-induced toxicity there is an increased generation of mitochondrial superoxide anion production, stimulated by impaired mitochondrial respiration and formation of adducts between the reactive metabolite NAPQI and sulfhydryls moieties from mitochondrial proteins (Yan et al., 2010)

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Autologous DFbs promote fibrosis of degenerative IVDs and maintain biomechanical properties Histological examinations of the IVDs were then carried out to assess the phenotypic changes following autologous DFb injection