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As an essential key coenzyme in all cellular metabolism, NAD+ participates in numerous vital physiological processes including energy metabolism, DNA damage repair and programmed apoptosis

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Loss of FeS cluster stability, often observed under oxidative stress, leads to the release of free iron into the mitochondrial matrix, exacerbating Fenton chemistry and fueling ferroptosis ( These findings highlight the pivotal role of mitochondrial dysfunction in ferroptosis-driven fibrosis

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[DOI] [PMC free article] [PubMed] [Google Scholar] 673.Wei H, Xiang L, Wayne AS, Chertov O, FitzGerald DJ, Bera TK, et al