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The c-MET serves as an attractive therapeutic target in SCLC, as shown through small interfering RNA (siRNA) and selective prototype c-MET inhibitor SU11274, inhibiting the phosphorylation of c-MET itself and its downstream molecules such as AKT, S6 kinase, and ERK1/2

Any pharmacokinetic differences relate to dissolution rate and absorption site pH dynamics rather than inherent compound differences

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[ 3 H]BPC157 was rapidly metabolized into a variety of small peptide fragments in vivo , thus forming single amino acids that entered normal amino acid metabolism and excretion pathways

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