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These findings support the notion from the imatinib-treated tumors that ablation of perivascular cells by PDGFR inhibitors allowed excessive sprouting of the tumor vasculatures

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These observations also highlight that therapeutic approaches should be directed at specific aspects of astrocyte reactivity, for example astrocyte Ccl2 production, rather than at attenuating reactivity per se, which may lead to unexpected and undesirable consequences

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