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Disruptions in cellular homeostasis contribute to chronic inflammation, immune dysfunction, metabolic dysfunction, telomere shortening, and accelerated aging

We previously discovered that, regardless of its activation state, D2R can enhance the membrane expression of DAT and consequently DAT-mediated reuptake of dopamine through their direct protein-protein interaction [23]

Additionally, the different patterns of hemolysis are the key point for differentiation, despite both conditions sharing a biochemical profile (elevated LDH, low haptoglobin, schistocytosis)