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105 The pathogenesis of AD may depend on the reduced activity and different polymorphisms of the paraoxonase-1 (PON1) enzyme, which accumulates in the body as a result of long-term exposure to OP pesticides, and the reduction of PON1 activity leads to an increase in activity against OP pesticides, which consequently causes symptoms of dementia, memory impairment, and other AD symptoms

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Although the blood urea level may be transiently elevated, the development of renal failure in children is exceedingly rare, even in the presence of massive hemoglobinuria (see Fig