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ghk-cu superoxide dismutase

ghk-cu superoxide dismutase Transduction of jellyfish mediated by TAT peptide ameliorates H2O2-induced oxidative stress in HaCaT cells Superoxide dismutase and neurological disorders

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Description

J Immunol (2011) 186:717686

ghk-cu superoxide dismutase Transduction of jellyfish mediated by TAT peptide ameliorates H2O2-induced oxidative stress in HaCaT cells Superoxide dismutase and neurological disorders

TB-500 (a Thymosin Beta-4 fragment) acts as a G-actin sequestering peptide affecting cytoskeletal remodelling and cell migration, with effects measured at the actin polymerization level rather than at receptor-pathway level

ghk-cu superoxide dismutase Transduction of jellyfish mediated by TAT peptide ameliorates H2O2-induced oxidative stress in HaCaT cells Superoxide dismutase and neurological disorders

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ghk-cu superoxide dismutase Transduction of jellyfish mediated by TAT peptide ameliorates H2O2-induced oxidative stress in HaCaT cells Superoxide dismutase and neurological disorders

Gerke, O.Q

ghk-cu superoxide dismutase Transduction of jellyfish mediated by TAT peptide ameliorates H2O2-induced oxidative stress in HaCaT cells Superoxide dismutase and neurological disorders

Primers used in this study are listed in Table 1, and when necessary were designed using mRNA sequences obtained from Entrez (www.ncbi.nlm.nih.gov) and Primer3 [36], and then verified using BLAST (www.ncbi.nlm.nih.gov)

ghk-cu superoxide dismutase Transduction of jellyfish mediated by TAT peptide ameliorates H2O2-induced oxidative stress in HaCaT cells Superoxide dismutase and neurological disorders
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