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High SF-1 expression in steroidogenic tissue likely contributes this preponderance of FDXR, as SF-1-binding sites are present in the FDXR promoter and SF-1 overexpression in adrenal cell models drives FDXR expression (85)

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b , SDS-PAGE immunoblot of electron transport chain proteins in isolated WAT mitochondria from Control and Bmal1-KO mice harvested at the indicated timepoints (n = 6)

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