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To explore the possible molecular mechanisms linking inflammation and iron overload in OA, they used IL-1 or TNF- to treat chondrocytes and observed increased TfR1 and DMT1 but decreased FPN, which could lead to intracellular iron accumulation (42)

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doi: 10.1007/s00284-019-01813-6 14

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Wang et al., 2017[43])

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