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Interestingly, it has been previously reported that LMP2A enhances MYC driven lymphomagenesis through activation of the PI3K-pathway (181183), suggesting that activation of PI3K by LMP2A might be an alternative and/or convergent mechanism to the one driven by TCF3 / ID3 mutations

This allows each cell to produce exactly what is needed to restore its specific redox balance without risking the over-suppression of beneficial, low-level ROS signaling that is absolutely necessary for normal cellular function and immune defense

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doi: 10.1016/j.preteyeres.2007.01.004 98