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KPV, derived from the C-terminal region of alpha-melanocyte-stimulating hormone, modulates NF-B and cytokine signaling

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HDAC3 and HDAC11, for instance, regulate oxidative stress responses and A metabolism, whereas HDAC7 contributes to tau pathology by suppressing lysosomal biogenesis through transcription factor EB (TFEB) deacetylation and by enhancing inflammatory signaling via the IKKNF-B pathway 31