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The KD modifies protein utilization, as many proteins are taken up by the body for the process of gluconeogenesis while the remainder contribute to tissue repair [2]

While most previous treatments worked by reducing platelet destruction through a general immune system effect, newer medicines target the pathophysiology of ITP more precisely by increasing platelet production or decreasing platelet destruction

2026 Feb 28;24(1):315

Despite significant progress in grasping the role of probiotics, hurdles remain such as strain specificity, limited clinical data and environmental factors affecting probiotic efficacy

We have previously demonstrated that ArfGAP1 serves as a robust substrate of LRRK2-mediated phosphorylation by in vitro radioactive kinase assay [16]