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This pathway, termed TMED10-channeled unconventional protein secretion (THU), relies on the ERGIC-localized TMED10 protein to facilitate the entry of IL1 family factors into the secretory system, initiating unconventional secretion 17

While Lip-1 alleviated the lethality of DEM (Figure 1E), it did not prevent the fall in glutathione that was induced by aging (as assayed on Day 4) or by DEM (Figure 1F)

Ive had Crohns disease for 13 years, and in that time, Ive experienced a variety of extraintestinal manifestations and side effects of living with this disease, but arguably none were more debilitating than my experience with a vitamin B-12 deficiency

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