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Preclinical investigations consistently demonstrate that luteolin may mitigate amyloid-beta (A)-induced neurotoxicity, inhibits tau hyperphosphorylation, modulates neuroinflammation driven by microglia, and enhances cognitive functions such as learning and memory in models of AD ( Although clinical evidence remains somewhat sparse, it further emphasizes the critical importance of examining luteolin within the context of AD

Key Points: Report 96365 for the initial hour of IV infusion, not for IV push or hydration

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Following PTZ treatment, there was a significant increase in the expression of iron regulatory protein (IRP) 1 (IRP1), IRP2, and TfR1 in mice, while FPN1 and FTH1 showed significant reductions