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35 The prospect of personalized medicine, tailoring treatment plans based on individual genetic and metabolic profiles, 54 holds promise for more effective and targeted management in the future

88 This conclusion was further supported by the demonstration of a dramatic increase in nitrotyrosine staining and liver injury in MnSOD (SOD2) partial knockout mice, 90 as well as the elimination of nitrotyrosine protein adducts and liver injury in animals treated with mitochondrial-targeted SOD mimetics such as Mito-TEMPO 91 and Mito-Q

11 , e1004870 (2015)

Variants in the last two groups, although of much interest to biologists, geneticists, and anthropologists, are of no major clinical significance

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