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In HFD-fed mice, the treatment demonstrated improvements in glucose tolerance, reduced hepatic steatosis, and alleviated adipose inflammation, partially by restoring oxidative stress, inflammation, and ERSrelated gene expression through SIRT1 (Yoshino et al

A.CantaA.SalaB.ColomboM.et al (2014)

By investigating the underlying mechanisms and potential clinical implications, the present review highlights that regulation of cuproptosis provides a promising approach for modulating disease progression and developing personalized treatment strategies for AD

In addition, antioxidants have also been shown to be capable of reversing certain altered biochemical variables and this ability enhances its potential to provide for optimal therapeutic benefits while causing fewer or minimal side effects