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This action protects alveolar epithelial cells against inflammation, injury, and apoptosis via the repression of prolyl hydroxylase 2/HIF-1/MAPK signaling, suggesting its potential in treating COPD and pulmonary fibrosis (Guan et al., 2020)

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At the molecular level, AHK-Cu also increases VEGF (vascular endothelial growth factor) production to improve scalp blood flow, and upregulates SOD (superoxide dismutase) to reduce the oxidative stress that triggers DHT-driven follicle miniaturization one of the primary mechanisms behind androgenetic alopecia

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Patients were categorized by ACLD status and AFP levels, with optimal cutoffs determined by the Youden Index

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Specific role of the extracellular signal-regulated kinase pathway in angiotensin II-induced cardiac hypertrophy in vitro

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Safety In l-ARG and placebo groups, no significant changes were detected from T0 to T1, regarding clinical and biochemical parameters, except for HDL cholesterol levels, which were significantly increased in l-ARG group ( p = 0.001) and decreased in placebo group ( p = 0.0001)

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