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Toll-like receptors, DAMPs, and IFNs have been implicated in RIC in brain ischemia (Hess et al., 2015) and the accumulating evidence on the key roles of classical CKs, ACKs, and MIF proteins, as discussed in this review article, suggest that some of these mediators, while being interesting therapeutic targets by themselves, could also be interesting in the context of RIC (Hamner et al., 2015

Samarasinghe, K

This process increases the expression of antioxidant genes, thereby bolstering the endogenous antioxidant capacity

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