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In our screening, the isoflavone (e.g., daidzein, genistein, and glycitein) and methoxyflavone (e.g., tricin, tangeretin, and diosmetin) derivatives as well as flavone O- and C-glycosides (e.g., isoquercitrin, orientin, and vitexin) were either completely inactive or exerted low inhibition capacities against both ChEs

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Glucose deprivation together with glycolysis inhibition (using 2-deoxy-glucose, 2-DG) resulted in the inhibition of NET formation (compared to untreated PMA-stimulated neutrophils), while the addition of glucose alone maintained NET formation in the same conditions (Rodrguez-Espinosa et al., 2015) (Table 2)

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Food-grade TiO2 impairs intestinal and systemic immune homeostasis, initiates preneoplastic lesions and promotes aberrant crypt development in the rat colon