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Description

Deng, H., Wu, S

In the two full-length constructs Sec was mutated either to Cys or Ser to enable high-yield heterologous overexpression in E

This downregulates small guanosine triphosphatase (GTPase) prenylation (e.g., RhoA activation), which otherwise suppresses eNOS expression and promotes EC inflammation [243,244,245,246]