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The PTGES-AKT-HIF-1 axis constitutes a core pathway for inflammation-induced glycolysis: pro-inflammatory factors such as IL-4 and IL-13 upregulate PTGES, which catalyzes the conversion of PGH2 to PGE2 to activate the PI3K-AKT pathway downstream of G protein-coupled receptors (32)

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However, high doses of vitamin E (800 mg/day) inhibit the symptoms progression and may even reverse some neurological symptoms [45]

These abnormally expressed thyrocytes function as antigen-presenting cells (APCs) that can present thyroid autoantigens to trigger autoimmune thyroid disease