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Hyperaldosteronism is associated with impaired vascular reactivity, and it has been shown that aldosterone induces a G6PD-deficient phenotype to impair endothelial function, and aldosterone antagonism or gene transfer of G6PD improves vascular reactivity [9]

In this study, we investigated the effects of dysregulated glutathione homeostasis, a principal feature of oxidative stress, on TNF-induced hepatotoxicity and its mechanistic implications in NAFLD progression

Oxidative stress increases during the aging process, resulting in either enhanced ROS generation or diminished antioxidant safeguards

reported that lower levels of TNF- and IL-8 could be observed after 5 weeks of IF (21)

Cell 176 , 1142 (2019)