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Mechanistically, HIF-1 binds to hypoxia-response elements (HREs) within the PD-L1 promoter, enhancing its expression in both tumor cells and immunosuppressive myeloid populations such as macrophages and MDSCs, thereby reinforcing an immune-resistant TME [329]

10.1093/nar/28.1.235 9 BrooksD

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As our understanding of the role of mTOR in cellular metabolism, immune regulation, and disease progression deepens, it becomes increasingly clear that future therapies must strike a delicate balance, sufficiently inhibiting pathological mTOR activity while preserving its essential physiological functions

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