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Therefore, the interaction involving the FOXO4-DRI peptide may host the potential to restore p53's ability to initiate the destruction of senescent cells through apoptosis

Mitochondrial SIRT is also capable of activating the AMPK signaling pathway, increasing autophagic function, and accelerating the clearance of the -Syn protein

Hence, it can be concluded that the exogenous GSH indeed leads to the increase in endogenous GSH, key to regulation of the AsA-GSH cycle, which oversees detoxification of ROS species, alleviating any cold stress-induced damage

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